Journal of Toxicology / 2015 / Article / Fig 3

Research Article

Increased Susceptibility to Ethylmercury-Induced Mitochondrial Dysfunction in a Subset of Autism Lymphoblastoid Cell Lines

Figure 3

AD LCLs demonstrate abnormal mitochondrial function at baseline and following ethylmercury exposure. (a) ATP-linked respiration, (b) proton leak respiration, (c) maximal respiratory capacity, and (d) reserve capacity are all overall significantly higher in the AD LCLs as compared to control LCLs. ATP-linked respiration is higher in the AD LCLs at every concentration of ethylmercury, while maximal respiratory capacity and reserve capacity are higher in the AD LCLs at every concentration except the highest concentration. ; ; ; ; indicates an overall statistical difference between LCL groups when differences at individual concentrations are not significant.

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