Mechanisms of L-Triiodothyronine-Induced Inhibition of Synaptosomal Na+-K+-ATPase Activity in Young Adult Rat Brain Cerebral Cortex
Figure 4
Modulation of the T3 action on synaptosomal Na+-K+-ATPase activity by a selective -adrenergic agonist (PHE) and a selective -antagonist (PRA) in vitro. A half-maximally effective dose of T3 (1 nM) was chosen from the dose-response curve for T3 in Figure 1. The doses for PHE and PRA used for the in vitro experiment were 1 nM in each case. The data are represented as mean ± SEM of five separate experiments, taking six animals in each group. The vertical lines denote SEM.
