Abstract
Recently, it has been reported that TLR2 on macrophages plays a unique
role in the inflammatory response and host defense to infection
with Borrelia burgdorferi (Bb) which is an etiologic agent of
Lyme disease. Experimental studies show that PMNs also play an
essential role in infection control by Bb. However,
there is no available data about TLR2 expression on PMN in the
course of Lyme disease. In the present study, TLR2 expression and
production of IL-1