Abstract
Aim. The aim of this study was to evaluate the presence of the cytokines tumor necrosis
factor-alpha (TNF-
Aim. The aim of this study was to evaluate the presence of the cytokines tumor necrosis
factor-alpha (TNF-
B. W. Neville, D. D. Damm, C. M. Allen, and J. R. Bouquort, Oral and Maxillofacial Pathology, W. B. Saunders, Philadelphia, Pa, USA, 4th edition, 1995.
C.-P. Chen, M. Hertzberg, Y. Jiang, and D. T. Graves, “Interleukin-1 and tumor necrosis factor receptor signaling is not required for bacteria-induced osteoclastogenesis and bone loss but is essential for protecting the host from a mixed anaerobic infection,” American Journal of Pathology, vol. 155, no. 6, pp. 2145–2152, 1999.
View at: Google ScholarK. Balto, H. Sasaki, and P. Stashenko, “Interleukin-6 deficiency increases inflammatory bone destruction,” Infection and Immunity, vol. 69, no. 2, pp. 744–750, 2001.
View at: Publisher Site | Google ScholarS. Yamano, J. C. Atkinson, B. J. Baum, and P. C. Fox, “Salivary gland cytokine expression in NOD and normal BALB/c mice,” Clinical Immunology, vol. 92, no. 3, pp. 265–275, 1999.
View at: Publisher Site | Google ScholarT. Radics, C. Kiss, I. Tar, and I. J. Márton, “Interleukin-6 and granulocyte-macrophage colony-stimulating factor in apical periodontitis: correlation with clinical and histologic findings of the involved teeth,” Oral Microbiology and Immunology, vol. 18, no. 1, pp. 9–13, 2003.
View at: Publisher Site | Google ScholarR. Vernal, A. Dezerega, N. Dutzan et al., “RANKL in human periapical granuloma: possible involvement in periapical bone destruction,” Oral Diseases, vol. 12, no. 3, pp. 283–289, 2006.
View at: Publisher Site | Google ScholarC. De Oliveira Rodini and V. S. Lara, “Study of the expression of macrophages and T cells in human granulomas and periapical cysts,” Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology, and Endodontics, vol. 92, no. 2, pp. 221–227, 2001.
View at: Publisher Site | Google ScholarD. T. Graves, C.-P. Chen, C. Douville, and Y. Jiang, “Interleukin-1 receptor signaling rather than that of tumor necrosis factor is critical in protecting the host from the severe consequences of a polymicrobe anaerobic infection,” Infection and Immunity, vol. 68, no. 8, pp. 4746–4751, 2000.
View at: Publisher Site | Google ScholarT. A. Silva, G. P. Garlet, V. S. Lara, W. Martins Jr., J. S. Silva, and F. Q. Cunha, “Differential expression of chemokines and chemokine receptors in inflammatory periapical diseases,” Oral Microbiology and Immunology, vol. 20, no. 5, pp. 310–316, 2005.
View at: Publisher Site | Google ScholarJ. Danin, L. E. Linder, G. Lundqvist, and L. Andersson, “Tumor necrosis factor-alpha and transforming growth factor-beta1 in chronic periapical lesions,” Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology, and Endodontics, vol. 90, no. 4, pp. 514–517, 2000.
View at: Publisher Site | Google ScholarR. A. Barkhordar, C. Hayashi, and M. Z. Hussain, “Detection of interleukin-6 in human dental pulp and periapical lesions,” Endodontics and Dental Traumatology, vol. 15, no. 1, pp. 26–27, 1999.
View at: Google ScholarF. Y. Bozkurt, E. Berker, S. Akkuş, and Ş. Bulut, “Relationship between interleukin-6 levels in gingival crevicular fluid and periodontal status in patients with rheumatoid arthritis and adult periodontitis,” Journal of Periodontology, vol. 71, no. 11, pp. 1756–1760, 2000.
View at: Publisher Site | Google ScholarD. Simčić, S. Pezelj-Ribarić, R. Gržić, J. Horvat, G. Brumini, and M. Muhvić-Urek, “Detection of salivary interleukin 2 and interleukin 6 in patients with burning mouth syndrome,” Mediators of Inflammation, vol. 2006, Article ID 54632, 4 pages, 2006.
View at: Publisher Site | Google ScholarR. F. De Jongh, K. C. Vissers, T. F. Meert, L. H. D. J. Booij, C. S. De Deyne, and R. J. Heylen, “The role of interleukin-6 in nociception and pain,” Anesthesia and Analgesia, vol. 96, no. 4, pp. 1096–1103, 2003.
View at: Publisher Site | Google Scholar