AMP-Activated Protein Kinase Activation during Cardioplegia-Induced Hypoxia/Reoxygenation Injury Attenuates Cardiomyocytic Apoptosis via Reduction of Endoplasmic Reticulum Stress
Figure 1
The results of western blot analysis of AMPKα 2, AMPKα 2, and p-AMPKα and AMPK activities in different groups are shown. Tubulin was used as internal control. (Results are representative of at least 3 separate experiments. * compared with Group 1; H: hypoxia; R: reoxygenation; Met: metformin.)
