Anti-Inflammatory Mechanism of Polyunsaturated Fatty Acids in Helicobacter pylori-Infected Gastric Epithelial Cells
Figure 4
Effects of PUFAs on activation of ERK and JNK in H. pylori-infected AGS cells. (a) AGS cells were infected with H. pylori for the indicated time points (15, 30, 60, and 120 min). Phosphospecific and total forms of ERK and JNK were measured by western blot analysis. (b) AGS cells were pretreated with individual fatty acid (100 M) for 24 h and then cultured with H. pylori for 30 min.
