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Research Article
Mediators of Inflammation
Volume 2018, Article ID 9878120, 3 pages
https://doi.org/10.1155/2018/9878120
Corrigendum

Corrigendum to “Formononetin Administration Ameliorates Dextran Sulfate Sodium-Induced Acute Colitis by Inhibiting NLRP3 Inflammasome Signaling Pathway”

1Department of Gastroenterology, Affiliated Hospital of Yangzhou University, Yangzhou University, Yangzhou, China
2Laboratory of Gastroenterology, Affiliated Hospital of Yangzhou University, Yangzhou University, Yangzhou, China
3Department of Immunology, School of Medicine, Yangzhou University, Yangzhou, China

Correspondence should be addressed to Yanbing Ding; nc.ude.uzy@gnidby

Received 30 July 2018; Accepted 31 July 2018; Published 6 September 2018

Copyright © 2018 Dacheng Wu et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.


In the article titled “Formononetin Administration Ameliorates Dextran Sulfate Sodium-Induced Acute Colitis by Inhibiting NLRP3 Inflammasome Signaling Pathway” [1], the protein labels in Figures 6(h) and 6(i) were reversed and should be corrected as follows:

Figure 6: NLRP3 inhibitor MCC950 eliminated the protective effect of H-For on acute colitis in mice. (a) The experimental protocol with For and MCC950 in an acute colitis model. (b) Body weights of mice and (c) disease activity index (DAI) during the disease process. (d) Morphological changes in the mouse colons, (e) variations of colon length of mice, (f) representative HE staining, and (g) histological scores of colonic tissue. (h) Protein levels of claudin-1, occludin, and ZO-1 and (i) NLRP3, ASC, and IL-1β were analyzed by western blotting. , , .

References

  1. D. Wu, K. Wu, Q. Zhu et al., “Formononetin administration ameliorates dextran sulfate sodium-induced acute colitis by inhibiting NLRP3 inflammasome signaling pathway,” Mediators of Inflammation, vol. 2018, Article ID 3048532, 12 pages, 2018. View at Publisher · View at Google Scholar · View at Scopus