Inflammation is common for heart disease and stroke patients and is thought to be a sign or atherogenic response. It is important to know what inflammation is and what it can do to the heart. Inflammation is most likely the ultimate cause of heart disease, and all factors such as genetic predisposition, activity level, and psychological stress are mediated by inflammation. Lifestyle and even genetic factor exert much of their influence on heart disease via their effects on inflammation.

We invite authors to submit review and/or original research articles that seek to define the role of innate and adaptive immunity in organ-specific adiposity with cardiovascular risk. Research areas involve ischemic heart disease, heart failure, atherosclerosis, and other research topics associated with organ-specific adiposity. Potential topics include, but are not limited to:

• Role of innate and adaptive immunity in organ-specific adiposity
• The contribution of specific immune cells in heart diseases
• Cardiovascular imaging of inflammation
• Novel in vivo and in vitro models describing the role of epicardial adipose tissue in cardiovascular inflammation and atherosclerosis
• The role of adipokines in heart diseases

Before submission authors should carefully read over the journal’s Author Guidelines, which are located at http://www.hindawi.com/journals/mi/guidelines/. Prospective authors should submit an electronic copy of their complete manuscript through the journal Manuscript Tracking System at http://mts.hindawi.com/submit/journals/mi/iii/ according to the following time table: