Active Calcium/Calmodulin-Dependent Protein Kinase II (CaMKII) Regulates NMDA Receptor Mediated Postischemic Long-Term Potentiation (i-LTP) by Promoting the Interaction between CaMKII and NMDA Receptors in Ischemia

Figure 3

(a) OGD for 10 min caused a significant increase in CaMKII activity (1.43±0.13, 𝑛=4, 𝑃<0.05). (b) OGD treatment for 25 min led to inhibition of CaMKII activity (0.65±0.07, 𝑛=3, 𝑃<0.05). (c) 1 hr after PT, CaMKII activity displayed an upward trend (1.38±0.07, 𝑛=3, 𝑃<0.05). (d) 12 hr after PT, CaMKII activity was inhibited (0.41±0.19, 𝑛=3, 𝑃<0.05).

(a)

(b)

(c)

(d)