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Neural Plasticity
Volume 2017, Article ID 3270725, 12 pages
https://doi.org/10.1155/2017/3270725
Research Article

Pathological Role of Peptidyl-Prolyl Isomerase Pin1 in the Disruption of Synaptic Plasticity in Alzheimer’s Disease

1Jiangsu Key Laboratory for Functional Substance of Chinese Medicine, Department of Biopharmaceutics and Food Science, School of Pharmacy, Nanjing University of Chinese Medicine, Nanjing 210023, China
2Department of Neurology, Drexel University College of Medicine, Philadelphia, PA 19102, USA
3Department of Oncology, Mayo Clinic, Rochester, MN 55905, USA
4Sbarro Institute for Cancer Research and Molecular Medicine, Center for Biotechnology, Temple University, Philadelphia, PA 19122, USA
5Department of Molecular Science and Nanosystems, Ca’ Foscari Università di Venezia, Via Torino 155, 30172 Venezia-Mestre, Italy
6Department of Medicinal Chemistry, China Pharmaceutical University, Nanjing 21009, China
7Department of Physiology, University of Pennsylvania School of Medicine, Philadelphia, PA 19104, USA

Correspondence should be addressed to Carol F. Lippa; ude.demlexerd@appil.lorac and Yuesong Gong; nc.ude.mcujn@gnogy

Received 28 October 2016; Accepted 12 December 2016; Published 26 March 2017

Academic Editor: Jason Huang

Copyright © 2017 Lingyan Xu et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

How to Cite this Article

Lingyan Xu, Zhiyun Ren, Frances E. Chow, et al., “Pathological Role of Peptidyl-Prolyl Isomerase Pin1 in the Disruption of Synaptic Plasticity in Alzheimer’s Disease,” Neural Plasticity, vol. 2017, Article ID 3270725, 12 pages, 2017. https://doi.org/10.1155/2017/3270725.