Pleiotropic Cellular Functions of PARP1 in Longevity and Aging: Genome Maintenance Meets Inflammation

<div class="floats-full-page floats-full-page-figure"><h2>Figure 4</h2><div class="floats-full-page-caption"><table>Simplified model of PARP1-dependent mechanisms of NF-<i>κ</i>B activation. For details see text. Scheme based on [<a href="../../321653/#B167">167</a>, <a href="../../321653/#B185">185</a>].</table></div><div class="floats-full-page-content"><table class="table-group" width="800px"><tr class="fig-image"><td><img alt="321653.fig.004" src="https://static-01.hindawi.com/articles/omcl/volume-2012/321653/figures/321653.fig.004.jpg" /></td></tr></table></div></div>

Figure 4 | Pleiotropic Cellular Functions of PARP1 in Longevity and Aging: Genome Maintenance Meets Inflammation

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Figure 4: Pleiotropic Cellular Functions of PARP1 in Longevity and Aging: Genome Maintenance Meets Inflammation

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<p><em>Oxidative Medicine and Cellular Longevity </em>publishes research involving cellular and molecular mechanisms of oxidative stress in the nervous system and related organ systems in relation to aging, immune function, vascular biology, metabolism etc.</p>


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<p>Chief Editor, Dr Vasquez-Vivar has experience in free radical and redox biology research including the discovery of the role of tetrahydrobiopterin in the regulation of superoxide generation by endothelial and neuronal nitric oxide synthase.</p>


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