Propofol Inhibits Lipopolysaccharide-Induced Tumor Necrosis Factor-Alpha Expression and Myocardial Depression through Decreasing the Generation of Superoxide Anion in Cardiomyocytes
Figure 6
In vivo cardiac function during endotoxemia.Wild type C57BL/6 mice were divided into four groups: control group, propofol alone group, LPS group, and LPS + propofol group. After 6 h, cardiac function was determined using a Millar pressure-conductance catheter. Changes in LV, (a); heart rate, (b); CO, (c); +dp/dt, (d) ESPVR were presented (each bar represents the mean ± S.D, compared with control group; compared with LPS group, ).
