[Retracted] PKCδ Promotes High Glucose Induced Renal Tubular Oxidative Damage via Regulating Activation and Translocation of p66Shc

Figure 6

PKCδ siRNA inhibits p66Shc translocation during HG treatment of HK-2 cells. HK-2 cells were transfected with PKCδ siRNA or scrambled siRNA and treated with HG for different time periods. Lysates of cytosolic and mitochondrial fractions were analyzed for p66Shc and p-p66Shc expression by Western blotting, and β-actin was used as a cytosolic loading control and Cox IV antibody was used as a mitochondrial loading control. Values are means ± SEM, . versus HG group.

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