CO<sub>2</sub> Pneumoperitoneum Preserves <i>β</i>-Arrestin 2 Content and Reduces High Mobility Group Box-1 (HMGB-1) Expression in  an Animal Model of Peritonitis

<table class="figure-group"><tr class="fig-image" id="a"><td><object data="https://static.hindawi.com/articles/omcl/volume-2015/160568/figures/160568.fig.005a.svgz" name="160568.fig.005a" type="image/svg+xml"></object></td></tr><tr class="fig-caption"><td><b>(a) </b>Naïve</td></tr><tr class="fig-image" id="b"><td><object data="https://static.hindawi.com/articles/omcl/volume-2015/160568/figures/160568.fig.005b.svgz" name="160568.fig.005b" type="image/svg+xml"></object></td></tr><tr class="fig-caption"><td><b>(b) </b>LT</td></tr><tr class="fig-image" id="c"><td><object data="https://static.hindawi.com/articles/omcl/volume-2015/160568/figures/160568.fig.005c.svgz" name="160568.fig.005c" type="image/svg+xml"></object></td></tr><tr class="fig-caption"><td><b>(c) </b>LS</td></tr></table>

<div>Immunohistochemical detection of myeloperoxidase in lung from naïve, LT, or LS animals. Samples collected from LT rats (b) show an increased inflammatory infiltrate and some grade of fibrosis of interalveolar septa compared to naïve tissue samples (a). In contrast, samples from animals subjected to CO<sub>2</sub> pneumoperitoneum revealed a reduced accumulation of inflammatory cells, as well as a substantial reduction of pulmonary fibrosis (c). Original magnification ×40.</div>

Oxidative Medicine and Cellular Longevity

fig5

Figure 5

Figure 5: CO<sub>2</sub> Pneumoperitoneum Preserves <i>β</i>-Arrestin 2 Content and Reduces High Mobility Group Box-1 (HMGB-1) Expression in  an Animal Model of Peritonitis 

Figure 5 | CO2 Pneumoperitoneum Preserves β-Arrestin 2 Content and Reduces High Mobility Group Box-1 (HMGB-1) Expression in  an Animal Model of Peritonitis 