Active Fragment of <i>Veronica ciliata</i> Fisch. Attenuates t-BHP-Induced Oxidative Stress Injury in HepG2 Cells through Antioxidant and Antiapoptosis Activities

<table class="table-group" id="tab1"><tr><td><table class="table"><tr><td class="thead-hr" colspan="4"><hr/></td></tr><tr class="thead"><td class="align_left" rowspan="2">Sample</td><td class="align_center" colspan="3">IC<sub>50</sub>(<i>μ</i>g/mL)</td></tr><tr class="thead"><td class="align_center">DPPH</td><td class="align_center">Superoxide anion</td><td class="align_center">Hydroxyl</td></tr><tr><td class="thead-hr" colspan="4"><hr/></td></tr><tr><td class="align_left">VCAF</td><td class="align_center">6.882 ± 0.83</td><td class="align_center">20.838 ± 1.319</td><td class="align_center">5.021 ± 0.701</td></tr><tr><td class="align_left">VC</td><td class="align_center">3.919 ± 0.593</td><td class="align_center">9.674 ± 0.986</td><td class="align_center">3.620 ± 0.559</td></tr><tr class="table-tr"><td colspan="4"><hr class="tbody-hr"/></td></tr></table></td></tr></table>

Antioxidant activity of VCAF (IC<sub>50</sub>, <i>μ</i>g/mL).

Oxidative Medicine and Cellular Longevity

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Table 1: Active Fragment of <i>Veronica ciliata</i> Fisch. Attenuates t-BHP-Induced Oxidative Stress Injury in HepG2 Cells through Antioxidant and Antiapoptosis Activities 

Table 1 | Active Fragment of Veronica ciliata Fisch. Attenuates t-BHP-Induced Oxidative Stress Injury in HepG2 Cells through Antioxidant and Antiapoptosis Activities 