Oxidative Medicine and Cellular Longevity / 2017 / Article / Fig 5

Research Article

Baicalin Ameliorates Experimental Liver Cholestasis in Mice by Modulation of Oxidative Stress, Inflammation, and NRF2 Transcription Factor

Figure 5

Baicalin improved BDL-induced decrease of mitochondrial membrane complex activities in mouse liver. BDL caused decrease in mitochondrial membrane complexes as measured from enzyme activities of electron transport chain complex I, complex II, and complex IV. BDL mediated decrease of mitochondrial complex activities were improved level by alternate day of baicalin treatment. Results are mean ± S.E.M. for each group. versus control and # versus BDL.

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