Inhibition of miR-302 Suppresses Hypoxia-Reoxygenation-Induced H9c2 Cardiomyocyte Death by Regulating Mcl-1 Expression

Figure 3

H9c2 cardiomyocytes subjected to H/R injury in the presence of different concentrations of nonsense control (NC), miR-302 mimic (m302), and/or miR-302 antagomir (i302). Mcl-1 expression was assessed by (a) real-time qPCR and (b) western blot analysis. β-actin was used as an internal control. Data are presented as the mean ± STD of six experiments. between two groups.

(a) Expression of Mcl-1 mRNA

(b) Expression of Mcl-1 protein