Protective Role of UCP2 in Oxidative Stress and Apoptosis during the Silent Phase of an Experimental Model of Epilepsy Induced by Pilocarpine

<table class="table-group" id="tab1"><tr><td><table class="table"><tr><td class="thead-hr" colspan="5"><hr/></td></tr><tr class="thead"><td class="align_left">Hippocampal formation</td><td class="align_center">Control (saline)</td><td class="align_center">Acute (5H)</td><td class="align_center">Silent (5D)</td><td class="align_center">Chronic (60D)</td></tr><tr><td class="thead-hr" colspan="5"><hr/></td></tr><tr><td class="align_left">CA1</td><td class="align_center">+</td><td class="align_center">+</td><td class="align_center">+++</td><td class="align_center">++</td></tr><tr><td class="align_left">CA3</td><td class="align_center">++</td><td class="align_center">+</td><td class="align_center">+++</td><td class="align_center">+</td></tr><tr><td class="align_left">Dentate gyrus</td><td class="align_center">+</td><td class="align_center">+</td><td class="align_center">+++</td><td class="align_center">++</td></tr><tr class="table-tr"><td colspan="5"><hr class="tbody-hr"/></td></tr></table></td></tr><tr class="table-fn"><td><div>Staining of phospho-AKT was scored by two independent observers as follows: +: low; ++: moderate; +++: high intensity based on photomicrographs presented in Figure <a href="../fig3/">3</a>. Control: saline-treated rats; acute: 5 hours after <i>status epilepticus</i> (SE); silent: 5 days after SE; chronic: 60 days after SE.<br/></div></td></tr></table>

<div>Immunoreactivity for phospho-AKT in the CA1, CA3, and dentate gyrus in an epilepsy model induced by pilocarpine administration.</div>

Oxidative Medicine and Cellular Longevity

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Table 1

Table 1: Protective Role of UCP2 in Oxidative Stress and Apoptosis during the Silent Phase of an Experimental Model of Epilepsy Induced by Pilocarpine 

Table 1 | Protective Role of UCP2 in Oxidative Stress and Apoptosis during the Silent Phase of an Experimental Model of Epilepsy Induced by Pilocarpine 