Histone Acetyltransferase-Dependent Pathways Mediate Upregulation of NADPH Oxidase 5 in Human Macrophages under Inflammatory Conditions: A Potential Mechanism of Reactive Oxygen Species Overproduction in Atherosclerosis

<div>Comparative reactivity analysis of two different anti-Nox5 antibodies in protein extracts derived from cultured human Mac. Representative immunoblots depicting the Nox5 protein expression in Mac employing sc-67006 antibody (a) or ab191010 antibody (b). Immunogen sequences of sc-67006 antibody (c) and ab191010 antibody (d).</div>

Oxidative Medicine and Cellular Longevity

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Figure 2: Histone Acetyltransferase-Dependent Pathways Mediate Upregulation of NADPH Oxidase 5 in Human Macrophages under Inflammatory Conditions: A Potential Mechanism of Reactive Oxygen Species Overproduction in Atherosclerosis 

Figure 2 | Histone Acetyltransferase-Dependent Pathways Mediate Upregulation of NADPH Oxidase 5 in Human Macrophages under Inflammatory Conditions: A Potential Mechanism of Reactive Oxygen Species Overproduction in Atherosclerosis 