Upregulation of Transient Receptor Potential Canonical Type 3 Channel via AT1R/TGF-<i>β</i>1/Smad2/3 Induces Atrial Fibrosis in Aging and Spontaneously Hypertensive Rats

<div>Atrial structural remodeling measured by H&amp;E staining from WKY 4m-o, WKY 24m-o, SHR 4m-o, and SHR 24m-o rats. Representative H&amp;E staining: (a) magnification ×4, (b) magnification ×400, and (c) magnified the blue grid from (b).</div>

Oxidative Medicine and Cellular Longevity

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Figure 1

Figure 1: Upregulation of Transient Receptor Potential Canonical Type 3 Channel via AT1R/TGF-<i>β</i>1/Smad2/3 Induces Atrial Fibrosis in Aging and Spontaneously Hypertensive Rats 

Figure 1 | Upregulation of Transient Receptor Potential Canonical Type 3 Channel via AT1R/TGF-β1/Smad2/3 Induces Atrial Fibrosis in Aging and Spontaneously Hypertensive Rats 