Hypoxia and Inflammation as a Consequence of <i>β</i>-Fibril Accumulation: A Perspective View for New Potential Therapeutic Targets

<div>Aspects of myocardial atrial amyloidosis with intracellular accumulation of <i>β</i>-fibrils constituted of polymerized ANP (atrial natriuretic factor) peptides. (a) An advanced disorganization of the cytoplasm is evident due to the displacement of the various cytoplasmic components. (b) High-magnification detail of 10 nm <i>β</i>-fibril aggregate.</div>

Oxidative Medicine and Cellular Longevity

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Figure 3: Hypoxia and Inflammation as a Consequence of <i>β</i>-Fibril Accumulation: A Perspective View for New Potential Therapeutic Targets 

Oxidative Medicine and Cellular Longevity

Hypoxia and Inflammation as a Consequence of β-Fibril Accumulation: A Perspective View for New Potential Therapeutic Targets

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