[Retracted] Proanthocyanidins Antagonize Arsenic-Induced Oxidative Damage and Promote Arsenic Methylation through Activation of the Nrf2 Signaling Pathway

<div>ROS changes in L-02 cells after arsenic and/or grape seed procyanidin extract treatment for 24 h. The more the number of FITC-A<sup>+</sup> cells, the more the ROS in the cells: (a1) the control group, (a2) the 10 mg/L GSPE group, (b1) the 25 mg/L GSPE group, (b2) the 50 mg/L GSPE group, (c1) the arsenic (25 <i>μ</i>M) group, (c2) the arsenic (25 <i>μ</i>M) + 10 mg/L GSPE group, (d1) the arsenic (25 <i>μ</i>M) + 25 mg/L GSPE group, and (d2) the arsenic (25 <i>μ</i>M) + 50 mg/L GSPE group.</div>

Oxidative Medicine and Cellular Longevity

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Figure 4

Figure 4: [Retracted] Proanthocyanidins Antagonize Arsenic-Induced Oxidative Damage and Promote Arsenic Methylation through Activation of the Nrf2 Signaling Pathway 

Figure 4 | [Retracted] Proanthocyanidins Antagonize Arsenic-Induced Oxidative Damage and Promote Arsenic Methylation through Activation of the Nrf2 Signaling Pathway 