Pharmic Activation of PKG2 Alleviates Diabetes-Induced Osteoblast Dysfunction by Suppressing PLC<i>β</i>1-Ca<sup>2+</sup>-Mediated Endoplasmic Reticulum Stress

<div>Schematic illustration of the detailed mechanisms responsible for cinaciguat provided a favorable effect on promoting osseointegration in T2DM.</div>

Oxidative Medicine and Cellular Longevity

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Figure 8

Figure 8: Pharmic Activation of PKG2 Alleviates Diabetes-Induced Osteoblast Dysfunction by Suppressing PLC<i>β</i>1-Ca<sup>2+</sup>-Mediated Endoplasmic Reticulum Stress 

Figure 8 | Pharmic Activation of PKG2 Alleviates Diabetes-Induced Osteoblast Dysfunction by Suppressing PLCβ1-Ca2+-Mediated Endoplasmic Reticulum Stress 