Synergistic Antiproliferative Effects of Combined γ-Tocotrienol and PPARγ Antagonist Treatment Are Mediated through PPARγ-Independent Mechanisms in Breast Cancer Cells
Figure 1
Treatment effects of γ-tocotrienol, PPARγ agonists, and PPARγ antagonists on +SA cells. +SA cells were plated at a density of 04 (6 wells per group) in 24-well culture plates and exposed to treatment media for a 4-day period. Afterwards viable cell number was determined using MTT colorimetric assay. Vertical bars indicate mean cell count ± SEM in each treatment group. * as compared with vehicle-treated controls.
