Figure 2: Interhemispheric competition following a stroke. The model suggests that the contralesional (unaffected) motor region exerts an excessive inhibitory influence on the ipsilesional (affected) motor cortex which might limitpoststroke motor recovery. The model provides a hypothetical framework for developing therapeutic strategies. (a) Upregulation of neural excitability of the intact regions of the ipsilesional (affected) motor cortex by anodal tDCS. (b) Downregulation of excitability of the contralesional (unaffected) motor cortex by cathodal tDCS.