and Receptors Contribute to Serotonin-Induced Vascular Dysfunction in Diabetes
Figure 5
Contractions of aorta from non-diabetic (a c, e, and g) and diabetic (b, d, f, and h) mice to serotonin, α-methyl-5HT, TCB-2, and norfenfluramine before (Control) and after inhibition of rho kinase with H1152 (1 μM). All data are mean ± SEM. versus Control.