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Mediators of Inflammation
Volume 2013 (2013), Article ID 691975, 2 pages
http://dx.doi.org/10.1155/2013/691975
Erratum

Erratum to “NF- B Signaling in the Brain of Autistic Subjects”

Department of Neurochemistry, New York State Institute for Basic Research in Developmental Disabilities, 1050 Forest Hill Road, Staten Island, New York, NY 10314, USA

Received 21 January 2013; Accepted 5 February 2013

Copyright © 2013 Mazhar Malik et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.


In the original paper, mistakes occurred in Figures 1, 2, and 3 should be replaced with the figures below. In addition, a new author Fujiang Cao has been added as the second author in this paper.

fig1
Figure 1: IKK protein expression in the cerebellum of autistic subjects. (a) Western blot studies on cerebellar homogenates using IKK antibody (dilution 1 : 1000). Lanes 1–7 represent control subjects and lanes 8–14 represent autistic subjects. (b) The blots shown in (a) were quantitated after being normalized by actin. Data are shown as mean SE. * versus control group.
fig2
Figure 2: I Bα inhibitory subunit expression in the cerebellum of autistic subjects. Western blot studies on cerebellar homogenates using I Bα antibody (dilution 1 : 1000). Lanes 1–7 represent control subjects and lanes 8–14 represent autistic subjects. The western blot results were quantitated after being normalized by actin. Data are shown as mean SE.
fig3
Figure 3: NF- B p65 protein expression in the cerebellum of autistic subjects. Western blot studies on cerebellar homogenates using I Bα antibody (dilution 1 : 500). Lanes 1–7 represent control subjects and lanes 8–14 represent autistic subjects. The western blot results were quantitated after being normalized by actin. Data are shown as mean SE.