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Tuberculosis Research and Treatment
Volume 2010, Article ID 517547, 8 pages
Research Article

Decreased Expression of T-Cell Costimulatory Molecule CD28 on CD4 and CD8 T Cells of Mexican Patients with Pulmonary Tuberculosis

1Facultad de Farmacia, Universidad Autonoma del Estado de Morelos, 62209 Morelos, Mexico
2Departamento de Inmunologia, Instituto de Investigaciones Biomedicas, Universidad Nacional Autonoma de Mexico, Apartado Postal 70228 Ciudad Universitaria, 04510, Mexico City, Mexico

Received 25 January 2010; Revised 1 July 2010; Accepted 19 July 2010

Academic Editor: Robert S. Wallis

Copyright ยฉ 2010 German Bernal-Fernandez et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

Citations to this Article [4 citations]

The following is the list of published articles that have cited the current article.

  • Marius Strioga, Vita Pasukoniene, and Dainius Characiejus, “CD8+ CD28− and CD8+ CD57+ T cells and their role in health and disease,” Immunology, vol. 134, no. 1, pp. 17–32, 2011. View at Publisher ยท View at Google Scholar
  • Devin Morris, Melissa Khurasany, Thien Nguyen, John Kim, Frederick Guilford, Rucha Mehta, Dennis Gray, Beatrice Saviola, and Vishwanath Venketaraman, “Glutathione and infection,” Biochimica et Biophysica Acta (BBA) - General Subjects, vol. 1830, no. 5, pp. 3329–3349, 2013. View at Publisher ยท View at Google Scholar
  • Fabrizio Cantini, Laura Niccoli, and Delia Goletti, “Tuberculosis risk in patients treated with non-anti-tumor necrosis Factor-a (TNF-a) targeted biologics and recently licensed TNF-a inhibitors: Data from clinical trials and national registries,” Journal of Rheumatology, vol. 41, no. 91, pp. 56–64, 2014. View at Publisher ยท View at Google Scholar
  • Fabrizio Cantini, Carlotta Nannini, Laura Niccoli, Linda Petrone, Giuseppe Ippolito, and Delia Goletti, “Risk of Tuberculosis Reactivation in Patients with Rheumatoid Arthritis, Ankylosing Spondylitis, and Psoriatic Arthritis Receiving Non-Anti-TNF-Targeted Biologics,” Mediators of Inflammation, vol. 2017, pp. 1–15, 2017. View at Publisher ยท View at Google Scholar