An Entropy-Based Model for Basal Ganglia Dysfunctions in Movement Disorders

Figure 1

This figure shows a hypothetical relation between neuronal entropy and the aptitude of the downstream of information to generate movement regarding the conditions of hypokinesia and hyperkinesia. Low neuronal complexity could result in a hyperlegible signals and increased motor production. In contrast, high neuronal complexity could result in an indecipherable signal reducing motor production. Anti-Parkinsonian treatments reduce both hypokinesia and complexity. Since the comparisons between movement disorders and control remain unestablished, the healthy condition is envisaged with an adequate degree of GPi complexity allowing the emergence of noticeable information related to voluntary movement.