Upregulation of Transglutaminase and <i>ε</i>
            (<i>γ</i>-Glutamyl)-Lysine in the Fisher-Lewis Rat Model of 
            Chronic Allograft Nephropathy

<table>A representative field is shown from terminal kidney tissue of a glomerulus (a), the tubulointerstitium (b), and an arteriole (c) from an isograft and a glomerulus (d), the tubulointerstitium (e), and an arteriole (f) from an allograft. Sections were stained with Masson’s trichrome and visualised using ×20 objective.</table>

BioMed Research International

fig3

Figure 3

Figure 3: Upregulation of Transglutaminase and <i>ε</i>
            (<i>γ</i>-Glutamyl)-Lysine in the Fisher-Lewis Rat Model of 
            Chronic Allograft Nephropathy 

Figure 3 | Upregulation of Transglutaminase and ε
            (γ-Glutamyl)-Lysine in the Fisher-Lewis Rat Model of 
            Chronic Allograft Nephropathy 

BioMed Research International

Upregulation of Transglutaminase and ε (γ-Glutamyl)-Lysine in the Fisher-Lewis Rat Model of Chronic Allograft Nephropathy

Figure 3