Review Article
Regulated Control of the Assembly and Diversity of LPS by Noncoding sRNAs
Table 1
Known and predicted sRNAs involved in LPS biosynthesis and its modifications.
| sRNA | Target | Effect on LPS | Reference(s) |
| GlmY/GlmZ | GlmS | GlcN6P precursor of LPS | [35, 39, 40] | MicA | Repression of PhoP/Q | P-EtN addition to the second Kdo | [21, 25] | ā | Repression of WaaR | Switch between glycoforms IV and V | [25] | ā | Repression of LpxT | Incorporation of lipid A modifications | [24] | GcvB | Repression of PhoP/Q | Direct effect on LPS not known | [68] | RybB | Repression of WaaR | Major control of the synthesis of glycoforms with truncation in the outer core and incorporation of the third Kdo | [25] | SlrA | Repression of Lpp | Levels of fatty acid (phospholipids vs LPS) control | [9, 22] | MgrR | Repression of EptB | Repression of P-EtN incorporation on the second Kdo | [25, 56] | ArcZ | Repression of EptB | Impact on LPS not known | [57] | MicF | LpxR | Deacylation of lipid A | [69] | GadY | waaQ operon | Overexpression causes transcriptional reduction, direct effect on transcription duo to base-pairing not know | [70] | RyeA | LpxM | Predicted from microarray data | [71] | RdlA | EptA | Predicted from microarray data | [71] | RyjB | RfaH | Predicted from microarray data | [71] |
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