Lost Polarization of Aquaporin4 and Dystroglycan in the Core Lesion after Traumatic Brain Injury Suggests Functional Divergence in Evolution

Figure 2

The abundance of AQP4, α-DG, and β-DG in the core lesion of brain cortex in WB. The left and right column, respectively, showed the expression of AQP4 (a–d) and DG (e–g) proteins in the lesion core of TBI group and the same area of control group. The picture (a, e) above and under the dash line represented TBI and control group, respectively. The molecular weight of β-actin, AQP4-M1, AQP4-M23, GAPDH, α-DG, and β-DG was 43, 34, 28–30, 37, 120, and 43 kD. TBI: traumatic brain injury group; Contr: the sham-operation group. . .