Clinical Study
The Impact of Vitamin D3 Supplementation on Mechanisms of Cell Calcium Signaling in Chronic Kidney Disease
Figure 2
The calcium entry through CRAC channels. (a) A typical experiment where CRAC channels were activated by intracellular Ca2+ depletion by Tg (1 μmol/L) and subsequently inhibited by 2APB (50 μmol/L). The representative trace was obtained from a single experiment from PBMCs of a CKD patient. (b) The comparison of the calcium entry through CRAC channels in PBMCs of CKD patients at baseline and after a 6-month vitamin D3 supplementation (, ).
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