Fast 3-Breath-Hold 3-Dimensional Tagging Cardiac Magnetic Resonance in Patients with Hypertrophic Myocardial Diseases: A Feasibility Study
Table 1
Circumferential Strain (%) Estimated by 3-Breath-Hold 3D Tagging CMR in Patients with Hypertrophic Myocardial Diseases and Normal Volunteers.
segment
1
2
3
4
5
6
7
8
9
10
11
12
13
14
15
16
EF (%)
HCM
−14.0
−20.0
28.0
−10.0
−22.0
23.0
6.8
−33.0
−25.0
−15.0
−25.0
17.0
−6.7
−24.0
−27.0
−23.0
62.5
−22.0
−15.0
−14.0
−7.9
−20.0
−11.0
−35.0
−13.0
−19.0
−23.0
−25.0
−26.0
−15.0
−25.0
−16.0
−25.0
79.7
−14.0
−22.0
−10.0
−13.0
−5.4
−14.0
−19.0
−14.0
−24.0
−12.0
−9.4
−14.0
−12.0
−15.0
−21.0
−27.0
67.1
−10.0
−22.0
6.0
−12.0
−22.0
−22.0
−12.0
−18.0
−23.0
−5.3
−16.0
−25.0
−14.0
−22.0
−31.0
−9.8
54.0
−16.8
−28.1
−13.6
−17.9
−21.5
−10.6
−22.6
−17.6
−16.9
−23.9
−31.7
−24.3
−35.7
−28.3
−18.2
−19.4
56.8
mean
−15.4
−21.4
−0.7
−12.2
−18.2
−6.9
−16.4
−19.1
−21.6
−15.8
−21.4
−14.5
−16.7
−22.9
−22.6
−20.8
64.0
HHD
−1.6
−17.0
−39.0
−6.5
−13.0
−1.6
−16.0
−9.9
−17.0
9.0
−4.2
−20.0
−6.7
−7.5
−12.0
−6.7
22.7
−10.0
−20.0
−8.6
−6.9
−5.3
−14.0
6.9
−9.2
−10.0
3.8
−4.0
−12.0
−5.2
−19.0
−12.0
−7.6
13.8
7.9
−14.1
−7.1
8.0
−9.4
−8.7
−14.0
−9.1
−18.0
−20.0
10.0
−16.0
−8.4
−12.0
−11.0
−6.9
33.5
Amyl
−22.0
−10.0
−11.0
−4.8
−10.0
−15.0
−18.8
−9.9
−17.6
−3.9
−18.5
−18.2
−13.5
−22.0
−10.2
−17.1
31.0
0.3
−7.6
0.6
−9.8
−2.4
−1.9
−1.9
−1.6
−14.0
−10.4
−3.0
−0.9
−0.7
−21.9
−12.7
−20.4
60.8
Normal
−14.3
−20.3
−15.4
−8.4
−12.9
−9.5
−9.2
−18.5
−23.5
−11.0
−6.0
−8.6
−25.5
−22.7
−17.7
−23.7
NA
Italic: abnormal circumferential strain (%), bold: segments with late gadolinium enhancement (LGE). HCM: hypertrophic cardiomyopathy, HHD: hypertensive heart disease, Amyl: cardiac amyloidosis, Normal: normal volunteers, NA: not available. The mean value of circumferential strain was shown in normal volunteers. In HCM, the segments with LGE were not identical to those with reduced strain in each patient. In HHD with low ejection fraction, the segments with reduced strain were observed more widely than those with LGE. The reduction in strain and LGE were observed diffusely in cardiac amyloidosis.
