BioMed Research International

Copyright © 2017 Piotr Surmiak et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

Linked References

1. H. Dolk, M. Loane, and E. Garne, “The prevalence of congenital anomalies in Europe,” Advances in Experimental Medicine and Biology, vol. 686, pp. 349–364, 2010. View at Publisher · View at Google Scholar · View at Scopus
2. A. Latos-Bielenska, A. Materna-Kiryluk, M. Badura-Stronka et al., “Wrodzone wady rozwojowe w Polsce w latach 2005-2006,” in Dane z Polskiego Rejestru Wrodzonych Wad Rozwojowych, Ośrodek Wydaw Nauk, Poznań, Poland, 2010. View at Google Scholar
3. C. L. Ulrey, L. Liu, L. G. Andrews, and T. O. Tollefsbol, “The impact of metabolism on DNA methylation,” Human Molecular Genetics, vol. 14, no. 1, pp. R139–R147, 2005. View at Publisher · View at Google Scholar · View at Scopus
4. H. Jakubowski, “Metabolism of homocysteine thiolactone in human cell cultures: Possible mechanism for pathological consequences of elevated homocysteine levels,” Journal of Biological Chemistry, vol. 272, no. 3, pp. 1935–1942, 1997. View at Google Scholar · View at Scopus
5. K. Livia, L. M. H. Michele, and A. P. S. Eric, “Congenital heart defects and maternal derangement of homocysteine metabolism,” The Journal of Pediatrics, vol. 135, no. 6, pp. 773-774, 1999. View at Publisher · View at Google Scholar
6. Y. Li, X. Chen, X. Zhao, and B. Hu, “Relationship between homocysteine induced apoptosis and teratogenesis in developing embryo,” Wei Sheng Yan Jiu, vol. 28, pp. 257-258, 1999. View at Google Scholar
7. R. P. M. Steegers-Theunissen, L. M. J. W. Van Driel, R. De Jonge et al., “Maternal global methylation status and risk of congenital heart diseases,” Obstetrics and Gynecology, vol. 112, no. 2, pp. 277–283, 2008. View at Publisher · View at Google Scholar · View at Scopus
8. C. A. Hobbs, M. A. Cleves, S. Melnyk, W. Zhao, and S. J. James, “Congenital heart defects and abnormal maternal biomarkers of methionine and homocysteine metabolism,” American Journal of Clinical Nutrition, vol. 81, no. 1, pp. 147–153, 2005. View at Google Scholar · View at Scopus
9. J. A. Greenberg, S. J. Bell, Y. Guan et al., “Folic acid supplementation and pregnancy: more than just neural tube defect prevention,” Reviews in Obstetrics and Gynecology, vol. 4, no. 2, pp. 52–59, 2011. View at Google Scholar
10. O. A. Ladipo, “Nutrition in pregnancy: mineral and vitamin supplements,” The American Journal of Clinical Nutrition, vol. 72, Supplement 1, pp. 280S–290S, 2000. View at Google Scholar
11. L. D. Botto, J. Mulinare, and J. D. Erickson, “Occurrence of congenital heart defects in relation to maternal multivitamin use,” American Journal of Epidemiology, vol. 151, no. 9, pp. 878–884, 2000. View at Publisher · View at Google Scholar · View at Scopus
12. MRC Vitamin Study Research Group, “Prevention of neural tube defects: results of the medical research council vitamin study,” The Lancet, vol. 338, no. 8760, pp. 131–137, 1991. View at Publisher · View at Google Scholar
13. T. J. Siddiqua, S. M. Ahmad, K. B. Ahsan et al., “Vitamin B12 supplementation during pregnancy and postpartum improves B12 status of both mothers and infants but vaccine response in mothers only: a randomized clinical trial in Bangladesh,” European Journal of Nutrition, vol. 55, no. 1, pp. 281–293, 2016. View at Publisher · View at Google Scholar · View at Scopus
14. DM. El-Abd, RN. Said, and BM. Hanna, “MTHFR C677T polymorphism is associated with certain subgroups of CHDs. Maternal folic acid supplementation proved protective against CHDs,” Comp Clin Pathol, vol. 23, p. 317, 2014. View at Publisher · View at Google Scholar
15. M. J. Boot, R. P. M. Steegers-Theunissen, R. E. Poelmann, L. Van Iperen, J. Lindemans, and A. C. Gittenberger-de Groot, “Folic acid and homocysteine affect neural crest and neuroepithelial cell outgrowth and differentiation in vitro,” Developmental Dynamics, vol. 227, no. 2, pp. 301–308, 2003. View at Publisher · View at Google Scholar · View at Scopus
16. S. K. Ratan, K. N. Rattan, R. M. Pandey et al., “Evaluation of the levels of folate, vitamin B12, homocysteine and fluoride in the parents and the affected neonates with neural tube defect and their matched controls,” Pediatric Surgery International, vol. 24, no. 7, pp. 803–808, 2008. View at Publisher · View at Google Scholar · View at Scopus
17. A. C. Verkleij-Hagoort, M. Verlinde, N. T. C. Ursem et al., “Maternal hyperhomocysteinaemia is a risk factor for congenital heart disease,” BJOG: An International Journal of Obstetrics and Gynaecology, vol. 113, no. 12, pp. 1412–1418, 2006. View at Publisher · View at Google Scholar · View at Scopus
18. J.-Y. Zhao, X.-Y. Yang, X.-H. Gong et al., “Functional variant in methionine synthase reductase intron-1 significantly increases the risk of congenital heart disease in the Han Chinese population,” Circulation, vol. 125, no. 3, pp. 482–490, 2012. View at Publisher · View at Google Scholar · View at Scopus
19. N. Solanky, A. R. Jimenez, S. W. D'Souza, C. P. Sibley, and J. D. Glazier, “Expression of folate transporters in human placenta and implications for homocysteine metabolism,” Placenta, vol. 31, no. 2, pp. 134–143, 2010. View at Publisher · View at Google Scholar · View at Scopus