Computational Analysis of Protein Structure Changes as a Result of Nondeletion Insertion Mutations in Human β-Globin Gene Suggests Possible Cause of β-Thalassemia

Conformation of model structures at 0ns, 10ns, 20ns, and 30ns. Cyan (Seq1), Magenta (Seq2), and Black brown (wild).

BioMed Research International

fig5

Figure 5

Figure 5: Computational Analysis of Protein Structure Changes as a Result of Nondeletion Insertion Mutations in Human β-Globin Gene Suggests Possible Cause of β-Thalassemia

Figure 5 | Computational Analysis of Protein Structure Changes as a Result of Nondeletion Insertion Mutations in Human β-Globin Gene Suggests Possible Cause of β-Thalassemia 