The Adenosine A2A Receptor Agonist Accelerates Bone Healing and Adjusts Treg/Th17 Cell Balance through Interleukin 6

Figure 10

A brief diagram of the mechanism of the adenosine A2A receptor agonist promoting tibial fracture healing in rats. The adenosine A2A receptor agonist enters the tissue through the fibrin gel sustained release system. This reduces IL-6 levels in the blood and local tissues of the fracture, thus promoting fracture healing and regulating the Treg and Th17 cells.