[Retracted] Activation of TREK1 Channel in the Anterior Cingulate Cortex Improves Neuropathic Pain in a Rat Model

Figure 4

The changes in pain threshold behavior of CCI rats and expression of TREK1 channel protein in ACC. The TREK1 channel was activated by LIN. Paw withdraw threshold value and Paw withdraw latency for Sham, CCI, and LIN groups after 1, 3, 5, 7, 14, and 21 days were detected. Treld positive rate, GFAP positive rate, NEUM positive rate, and current for three groups of rats were detected. The level of TERK1 protein in the ACC neurons was assessed by Western blot at 1, 3, 7, and 14 days.