Metabolic Alteration in Hepatocellular Carcinoma: Mechanism of Lipid Accumulation in Well-Differentiated Hepatocellular Carcinoma

Table 1

Clinical characteristics of the patients with HCC.

Number	103
Age (years)	68.1 ± 7.8
Gender (M/F)	79/24
Etiology
(HBV/HCV/HBV + HCV/NBNC)	15/65/2/21
Liver disease states
(Chronic hepatitis/liver cirrhosis)	57/46
Cancer cell differentiation
(Well-/moderately/poorly differentiated)	20/57/26
Paraclinical status
Alb (g/dl)	3.95 ± 0.44
T-Bil (mg/dl)	0.91 ± 0.58
AST (IU/l)	51.4 ± 41.8
ALT (IU/l)	44.9 ± 31.5
GGT (IU/l)	84.8 ± 96.9
ALP (IU/l)	317.3 ± 130.3
LDH (IU/l)	211.2 ± 71.4
TC (mg/dl)	168.9 ± 43.3
TG (mg/dl)	109.0 ± 56.6
FBS (mg/dl)	107.8 ± 42.9

Counts are presented as N; continuous data are presented as mean ± SD. Alb, albumin; T-Bil, total bilirubin; AST, aspartate aminotransferase; ALT, alanine aminotransferase; GGT, gamma-glutamyl transpeptidase; ALP, alkaline phosphatase; LDH, lactate dehydrogenase; TC, total cholesterol; TG, triglyceride; FBS, fasting blood sugar; HBV, hepatitis B virus; HCV, hepatitis C virus; NBNC, non-HBV and non-HCV.