- About this Journal ·
- Abstracting and Indexing ·
- Aims and Scope ·
- Article Processing Charges ·
- Articles in Press ·
- Author Guidelines ·
- Bibliographic Information ·
- Citations to this Journal ·
- Contact Information ·
- Editorial Board ·
- Editorial Workflow ·
- Free eTOC Alerts ·
- Publication Ethics ·
- Reviewers Acknowledgment ·
- Submit a Manuscript ·
- Subscription Information ·
- Table of Contents
Case Reports in Medicine
Volume 2011 (2011), Article ID 729862, 4 pages
Milk Alkali and Hydrochlorothiazide: A Case Report
Department of Internal Medicine, Meharry Medical College, 1818 Albion Street, Nashville, TN 37208, USA
Received 9 November 2010; Revised 12 February 2011; Accepted 28 March 2011
Academic Editor: Robert A. Kozol
Copyright © 2011 Babar Parvez et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
- B. W. Sippy, “Landmark article May 15, 1915: gastric and duodenal ulcer. Medical cure by an efficient removal of gastric juice corrosion. By Bertram W. Sippy,” Journal of the American Medical Association, vol. 250, no. 16, pp. 2192–2197, 1983.
- J. B. Kirsner and W. L. Palmer, “Alkalosis complicating the Sippy treatment of peptic ulcer,” Archives of Internal Medicine, vol. 69, pp. 789–807, 1942.
- L. L. Hardt and A. B. Rivers, “Toxic manifestations following the alkaline treatment of peptic ulcer,” Archives of Internal Medicine, vol. 31, pp. 171–180, 1923.
- C. L. Cope, “Base change in the alkalosis produced by the treatment of gastric ulcer with alkalies,” Clinical Science, vol. 2, pp. 287–300, 1936.
- C. H. Burnett, R. R. Commons, F. Albright, and J. E. Howard, “Hypercalcemia without hypercalciuria or hypophosphatemia, calcinosis and renal insufficiency,” The New England Journal of Medicine, vol. 240, pp. 787–794, 1949.
- S. Punsar and T. Somer, “The milk-alkali syndrome. A report of three illustrative cases and a review of the literature,” Acta Medica Scandinavica, vol. 173, pp. 435–449, 1963.
- D. E. McMillan and R. B. Freeman, “The milk alkali syndrome: a study of the acute disorder with comments on the development of the chronic condition,” Medicine, vol. 44, no. 6, pp. 485–501, 1965.
- R. E. Randall Jr., M. B. Strauss, and W. F. McNeely, “The milk-alkali synfcmme,” Archives of internal medicine, vol. 107, pp. 163–181, 1961.
- E. S. Orwoll, “The milk-alkali syndrome: current concepts,” Annals of Internal Medicine, vol. 97, no. 2, pp. 242–248, 1982.
- M. J. Brodie, P. C. Ganguli, A. Fine, and T. J. Thomson, “Effects of oral calcium gluconate on gastric acid secretion and serum gastrin concentration in man,” Gut, vol. 18, no. 2, pp. 111–114, 1977.
- J. Behar, M. Hitchings, and R. D. Smyth, “Calcium stimulation of gastrin and gastric acid secretion: effect of small doses of calcium carbonate,” Gut, vol. 18, no. 6, pp. 442–448, 1977.
- V. Matkovic, “Calcium metabolism and calcium requirements during skeletal modeling and consolidation of bone mass,” American Journal of Clinical Nutrition, vol. 54, no. 2, 1991.
- R. P. Heaney, S. Abrams, B. Dawson-Hughes et al., “Peak bone mass,” Osteoporosis International, vol. 11, no. 12, pp. 985–1009, 2000.
- S. C. Hebert, S. Cheng, and J. Geibel, “Functions and roles of the extracellular Ca2+-sensing receptor in the gastrointestinal tract,” Cell Calcium, vol. 35, no. 3, pp. 239–247, 2004.
- P. Ivanovich, H. Fellows, and C. Rich, “The absorption of calcium carbonate,” Annals of Internal Medicine, vol. 66, no. 5, pp. 917–923, 1967.
- B. A. Lamberg and B. Kuhlback, “Effect of chlorothiazide and hydrochlorothiazide on the excretion of calcium in urine,” Scandinavian Journal of Clinical and Laboratory Investigation, vol. 11, pp. 351–357, 1959.
- A. M. Parfitt, “Chlorothiazide-induced hypercalcemia in juvenile osteoporosis and hyperparathyroidism,” The New England Journal of Medicine, vol. 281, no. 2, pp. 55–59, 1969.
- A. M. Patel and S. Goldfarb, “Got calcium? Welcome to the calcium-alkali syndrome,” Journal of the American Society of Nephrology, vol. 21, no. 9, pp. 1440–1443, 2010.
- R. A. L. Sutton, N. L. M. Wong, and J. H. Dirks, “Effects of metabolic acidosis and alkalosis on sodium and calcium transport in the dog kidney,” Kidney International, vol. 15, no. 5, pp. 520–533, 1979.
- T. Nijenhuis, V. Vallon, A. W. C. M. Van Der Kemp, J. Loffing, J. G. J. Hoenderop, and R. J. M. Bindels, “Enhanced passive Ca2+ reabsorption and reduced Mg channel abundance explains thiazide-induced hypocalciuria and hypomagnesemia,” Journal of Clinical Investigation, vol. 115, no. 6, pp. 1651–1658, 2005.