Table of Contents Author Guidelines Submit a Manuscript
Case Reports in Neurological Medicine
Volume 2016, Article ID 5169056, 3 pages
http://dx.doi.org/10.1155/2016/5169056
Case Report

FLAIR Hyperintense Vessel Sign of Both MCAs with Severe Heart Failure

1Department of Neurology, Wonkwang University Sanbon Hospital, Sanbonro 321, Gunpo, Gyeonggi-do 15-865, Republic of Korea
2Department of Cardiology, Wonkwang University Sanbon Hospital, Sanbonro 321, Gunpo, Gyeonggi-do 15-865, Republic of Korea

Received 30 May 2016; Accepted 31 July 2016

Academic Editor: Norman S. Litofsky

Copyright © 2016 Donghee Kim et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

Abstract

Introduction. Fluid-attenuated inversion recovery hyperintense vessels (FHVs) can be seen in patients with occlusion or severe stenosis of the cerebral arteries. FHVs are known to reflect stagnant or slow blood flow within the cerebral artery. Case Report. A 75-year-old woman presented with suddenly developed gait disturbance. She had a history of hypertension, heart failure, and dementia. Brain MRI demonstrated FHVs within both middle cerebral arteries (MCAs). However, there was no acute ischemic lesion and severe stenosis or occlusion of the cerebral arteries. In the baseline routine laboratory investigations, the AST, ALT, and B-type natriuretic peptide levels were elevated. Transthoracic echocardiography (TTE) showed mitral valve prolapse with severe regurgitation. Blood pressure control and conservative management for ischemic hepatitis were performed. After 7 days, the transaminase levels were normalized, and the patient was able to walk with normal gait. Conclusions. In this patient, underlying chronic cerebral hypoperfusion and additionally decreased systemic perfusion seemed to provoke ischemic hepatitis and contribute to the development of FHVs.