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Case Reports in Neurological Medicine
Volume 2017, Article ID 2736402, 4 pages
Case Report

Watershed Infarct in Beta-Thalassemia Major Patient

1Isfahan Neurosciences Research Center, Alzahra Research Institute, Isfahan University of Medical Sciences, Isfahan, Iran
2Department of Neurology, Isfahan University of Medical Sciences, Isfahan, Iran
3Department of Neurology, Tehran University of Medical Sciences, Sina Hospital, Tehran, Iran

Correspondence should be addressed to Behnaz Ansari; moc.oohay@zanhebirasna

Received 11 August 2016; Revised 17 November 2016; Accepted 7 February 2017; Published 27 March 2017

Academic Editor: Dennis J. Rivet

Copyright © 2017 Behnaz Ansari et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.


Background. The mechanism of stroke in beta-thalassemia was reported previously as cardioembolic and hypercoagulable state. However, there is no report of watershed infarct in beta-thalassemia anemia. Method. We present an adult β-thalassemia major patient with manifest asymptomatic chronic left carotid occlusion who suffered watershed infarct. Result. In the presence of asymptomatic chronic left internal carotid occlusion, we assumed that severe anemia (hemoglobin = 3) at admission leads to watershed infarct. Conclusion. Watershed infarct seems to be the cause of stroke in cases of β-thalassemia major with severe anemia. Blood transfusion can be applied in the setting of acute brain ischemia in such high risk patients.