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Case Reports in Pediatrics
Volume 2016 (2016), Article ID 3034170, 2 pages
Case Report

Transient Creatine Kinase Elevation Followed by Hypocomplementemia in a Case of Rotavirus Myositis

Department of Pediatrics, Sendai Medical Center, 2-8-8 Miyagino, Miyagino-ku, Sendai, Miyagi 983-8520, Japan

Received 7 October 2015; Accepted 18 February 2016

Academic Editor: Josef Sykora

Copyright © 2016 Yuka Rokugo et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.


We report an infant case of rotavirus myositis, a rare complication of rotavirus infection. Complement levels of the patient were normal when serum creatine kinase (CK) level was at its peak and then decreased when the CK level became normalized. In a previous case report of rotavirus myositis, transient decrease of serum albumin, immunoglobulin, and complement levels was reported. The authors speculated that intravascular complement activation was caused by rotavirus and resulted in the pathogenesis of myositis, although complement levels at onset were not measured by the authors. In this report, however, we demonstrate that the complement activation of our patient is a result of, rather than the cause of, skeletal muscle damage.