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Case Reports in Urology
Volume 2014, Article ID 939268, 2 pages
Case Report

Fahr Syndrome Unknown Complication: Overactive Bladder

1Department of Urology, Faculty of Medicine, University of Kirikkale, Yahsihan, 71100 Kirikkale, Turkey
2Department of Neurology, Faculty of Medicine, University of Kirikkale, Turkey

Received 7 April 2014; Accepted 8 July 2014; Published 16 July 2014

Academic Editor: Bharat Rekhi

Copyright © 2014 Devrim Tuglu et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.


A 38-year-old male patient was admitted to our outpatient department because of frequency and urgency incontinence. During evaluation it was detected that the patient was suffering from frequency which was progressive for one year, feeling of incontinence, and urgency incontinence. There was no urologic pathology detected in patient’s medical and family history. Neurologic consultation was requested due to his history of boredom, reluctance to do business, balance disorders, and recession for about 3 years. Brain computerized tomography (CT) scan revealed that amorphous calcifications were detected in the bilaterally centrum semiovale, basal ganglia, capsula interna, thalami, mesencephalon, pons and bulbus, and the bilateral cerebellar hemispheres. We have detected spontaneous neurogenic detrusor overactivity without sphincter dyssynergia after evaluating the voiding diary, cystometry, and pressure flow study. We consider the detrusor overactivity which occurred one year after the start of the neurological symptoms as the suprapontine inhibition and damage in the axonal pathways in the Fahr syndrome.