Molecular Mechanisms in Exercise-Induced Cardioprotection

<table class="table-group" id="tab3"><tr><td><table class="table"><tr><td class="thead-hr" colspan="2"><hr/></td></tr><tr class="thead"><td align="left">Mechanisms which mainly decrease ROS production</td><td align="left">Mechanisms which mainly repair cellular damages</td></tr><tr><td class="thead-hr" colspan="2"><hr/></td></tr><tr><td align="left">(i) Sarco K<sub>ATP</sub> channles (and possibly mito K<sub>ATP</sub> channles)<br/>(ii) ↑NO<br/>(iii) Mitochondrial adaptations, including ↑MnSOD</td><td align="left">(i) HSPs<br/>(ii) ↑Autophagy<br/>(iii) ↑ER stress proteins</td></tr><tr class="table-tr"><td colspan="2"><hr class="tbody-hr"/></td></tr></table></td></tr></table>

Proposed mechanisms in exercise-induced cardioprotection.

Cardiology Research and Practice

tab3

Table 3

Table 3: Molecular Mechanisms in Exercise-Induced Cardioprotection 