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Evidence-Based Complementary and Alternative Medicine
Volume 2012, Article ID 895032, 9 pages
http://dx.doi.org/10.1155/2012/895032
Review Article

History and Mechanism for Treatment of Intracerebral Hemorrhage with Scalp Acupuncture

1The Third Clinical College of Zhejiang Chinese Medical University, Hangzhou 310005, China
2Department of Acupuncture and Moxibustion, General Hospital of the Chinese People’s Liberation Army, Beijing 100853, China
3Center of Neurology and Rehabilitation, The Second Affiliated Hospital of Wenzhou Medical College, Wenzhou 325027, China

Received 23 November 2011; Accepted 14 December 2011

Academic Editor: Gerhard Litscher

Copyright © 2012 Zhe Liu et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

Abstract

Intracerebral hemorrhage (ICH) is an important public health problem with high rates of mortality, morbidity, and disability, but no clinically proven treatment strategy is available to date. Scalp acupuncture (SA) refers to a therapy for treating diseases by needling and stimulating the specific areas of the scalp. The evidence from clinical studies suggested that SA therapy may produce significant benefits for patients with acute ICH. However, the therapeutic mechanisms are yet not well addressed. Therefore, in this paper, we provide a comprehensive overview on the history and mechanisms of SA therapy on acute ICH. Although SA has been practiced for thousands of years in China and could date back to 5 BC, SA therapy for acute ICH develops only in the recent 30 years. The possible mechanisms associated with the therapeutic effects of SA on ICH include the influence on hematoma, brain edema, and blood brain barrier, the products released from haematoma, the immune and inflammatory reaction, focal perihemorrhagic hypoperfusion and hemorheology, neuroelectrophysiology, and so on. At last, the existence of instant effect of SA on acute ICH and its possible mechanisms are presented.