Dietary Acacetin Reduces Airway Hyperresponsiveness and Eosinophil Infiltration by Modulating Eotaxin-1 and Th2 Cytokines in a Mouse Model of Asthma

<table>Acacetin suppressed eosinophil adhesion to BEAS-2B cells. HL-60 cells were treated with calcein-AM and added to BEAS-2B cells, and the results were observed using fluorescence microscopy. Note the adherence of HL-60 cells to normal (a) and TNF-<i>α</i>-activated BEAS-2B cells (b). Pretreatment of HL-60 cells with 3 <i>μ</i>M acacetin (c), 10 <i>μ</i>M acacetin (d), 30 <i>μ</i>M acacetin (e), and 100 <i>μ</i>M acacetin (f) reduced the adherence of HL-60 cells to BEAS-2B cells in a dose-dependent manner.</table>

Evidence-Based Complementary and Alternative Medicine

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Figure 8

Figure 8: Dietary Acacetin Reduces Airway Hyperresponsiveness and Eosinophil Infiltration by Modulating Eotaxin-1 and Th2 Cytokines in a Mouse Model of Asthma 

Figure 8 | Dietary Acacetin Reduces Airway Hyperresponsiveness and Eosinophil Infiltration by Modulating Eotaxin-1 and Th2 Cytokines in a Mouse Model of Asthma 