Salidroside Improves Homocysteine-Induced Endothelial Dysfunction by Reducing Oxidative Stress
The effect of homocysteine (Hcy) and salidroside (Sali) on the levels of p-eNOS and total eNOS in primary rat aortic endothelial cells. Homocysteine-induced reduction in p-eNOS was reversed by treatment with salidroside, tiron, and DPI. Results are means ± SEM of 4 separate experiments. * versus control; # versus Hcy.
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