Protective Effects of Salidroside on Mitochondrial Functions against Exertional Heat Stroke-Induced Organ Damage in the Rat

Figure 1

Salidroside suppressed EHS-induced morphologic alterations of mitochondria in cardiomyocytes and hepatocytes. Mitochondrial morphology in the EHS group and EHS + salidroside group (20, 50, and 100 μM) was observed by an electron microscope (15,000x). The red arrowheads indicated the mitochondrial swelling, myelin breakdown, and disappearance of cristae matrix granules and, in mitochondrial matrix or concentric cristae, indicated the electron-lucent areas formed.