Quercetin Improves Mitochondrial Function and Inflammation in H2O2-Induced Oxidative Stress Damage in the Gastric Mucosal Epithelial Cell by Regulating the PI3K/AKT Signaling Pathway

<div>Que alleviates H2O2-induced mitochondria dysfunction in GES-1 cells. (a) Flow cytometry was performed to measure the proportion of cells with decrease in MMP. (b) Western blot was performed to evaluate the phosphorylation of DRP1 and the expression of DRP1 and MFN2 in GES-1 cells. (c) The representative ultrastructure of mitochondria in GES-1 cells (12.0 K). Data presets as mean ± SD, n− = 3, ∗P &lt; 0.05 vs. control group, #P &lt; 0.05 vs. model group.</div>

Evidence-Based Complementary and Alternative Medicine

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Figure 2: Quercetin Improves Mitochondrial Function and Inflammation in H2O2-Induced Oxidative Stress Damage in the Gastric Mucosal Epithelial Cell by Regulating the PI3K/AKT Signaling Pathway

Figure 2 | Quercetin Improves Mitochondrial Function and Inflammation in H2O2-Induced Oxidative Stress Damage in the Gastric Mucosal Epithelial Cell by Regulating the PI3K/AKT Signaling Pathway 